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rabbit anti-rat p53  (AnaSpec)

 
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    AnaSpec rabbit anti-rat p53
    Rabbit Anti Rat P53, supplied by AnaSpec, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rabbit+anti-rat+p53/10__1177_slash_20587392211069771-30-0-15?v=AnaSpec
    Average 90 stars, based on 1 article reviews
    rabbit anti-rat p53 - by Bioz Stars, 2026-07
    90/100 stars

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    rabbit anti-rat p53  (AnaSpec)
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    AnaSpec rabbit anti-rat p53
    (A–E). Fluorescent immunohistochemical staining of (A) COX-2, (B) survivin, (C) <t>p53,</t> (D) connexin-43, and (E) Bcl-2. Photomicrographs of particular immunostaining of COX-2, p53, connexin-43, survivin, and Bcl-2 for fluorescence immunohistochemistry analyses shown by a greenish-yellow color and nuclear propidium iodide staining shown by a red color. Original magnification, 40×. Black arrows indicate immunopositive cells. Significant differences between Gp-I (control) and Gp-II (DSS) are shown by *** p < 0.001 and those between Gp-II and Gp-III are shown by the symbol # p < 0.05.
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    Image Search Results


    KEY RESOURCES TABLE

    Journal: Cell reports

    Article Title: PAD4 controls tumor immunity via restraining the MHC class II machinery in macrophages

    doi: 10.1016/j.celrep.2024.113942

    Figure Lengend Snippet: KEY RESOURCES TABLE

    Article Snippet: Rat monocloncal antibody anti-mouse TIM-4 (clone 54) , Cell Signaling Technology , Cat#12-5866-82; RRID: AB_1257163.

    Techniques: Recombinant, Plasmid Preparation, Lysis, Protease Inhibitor, SYBR Green Assay, Western Blot, Chromatin Immunoprecipitation, Magnetic Beads, Mutagenesis, Cell Isolation, Isolation, Transfection, Enzyme-linked Immunospot, Mass Spectrometry, Gene Expression, Transgenic Assay, CRISPR, Software

    Effect of treatment with HNPMI on apoptosis‐related gene and intracellular BCL‐2 and p53 protein expression. (a) Quantitative expression of apoptosis‐related genes including BAD, BAX, CASP, BCL‐2 and p53, represented as fold change (log 10 ) upon HNPMI treatment in HT‐29 and DLD‐1 cells. GAPDH was used as a loading control. Data shown are means ± SEM from n = 6 independent values. * P < 0.05, significantly different from control (zero log 10 change); two‐way ANOVA). The fold changes were normalized against the control. The arrows in the confocal microscopy images (63×) showing the expression and localization of (b) BCL‐2 and (c) p53 protein expression using the corresponding primary antibodies in both HT‐29 and DLD‐1 cells.

    Journal: British Journal of Pharmacology

    Article Title: A novel EGFR inhibitor, HNPMI, regulates apoptosis and oncogenesis by modulating BCL‐2/BAX and p53 in colon cancer

    doi: 10.1111/bph.16141

    Figure Lengend Snippet: Effect of treatment with HNPMI on apoptosis‐related gene and intracellular BCL‐2 and p53 protein expression. (a) Quantitative expression of apoptosis‐related genes including BAD, BAX, CASP, BCL‐2 and p53, represented as fold change (log 10 ) upon HNPMI treatment in HT‐29 and DLD‐1 cells. GAPDH was used as a loading control. Data shown are means ± SEM from n = 6 independent values. * P < 0.05, significantly different from control (zero log 10 change); two‐way ANOVA). The fold changes were normalized against the control. The arrows in the confocal microscopy images (63×) showing the expression and localization of (b) BCL‐2 and (c) p53 protein expression using the corresponding primary antibodies in both HT‐29 and DLD‐1 cells.

    Article Snippet: Two different primary antibodies were used, anti‐BCL‐2 (C‐2) mouse monoclonal IgG (Santa Cruz Biotechnology, Inc, Dallas, TX, USA, Cat# sc‐7382, RRID:AB_626736 ) and anti‐p53 rabbit polyclonal IgG (R&D Systems, Cat# AF1355, RRID:AB_354749 ).

    Techniques: Expressing, Control, Confocal Microscopy

    (A–E). Fluorescent immunohistochemical staining of (A) COX-2, (B) survivin, (C) p53, (D) connexin-43, and (E) Bcl-2. Photomicrographs of particular immunostaining of COX-2, p53, connexin-43, survivin, and Bcl-2 for fluorescence immunohistochemistry analyses shown by a greenish-yellow color and nuclear propidium iodide staining shown by a red color. Original magnification, 40×. Black arrows indicate immunopositive cells. Significant differences between Gp-I (control) and Gp-II (DSS) are shown by *** p < 0.001 and those between Gp-II and Gp-III are shown by the symbol # p < 0.05.

    Journal: ACS Omega

    Article Title: α-Terpineol Mitigates Dextran Sulfate Sodium-Induced Colitis in Rats by Attenuating Inflammation and Apoptosis

    doi: 10.1021/acsomega.3c04317

    Figure Lengend Snippet: (A–E). Fluorescent immunohistochemical staining of (A) COX-2, (B) survivin, (C) p53, (D) connexin-43, and (E) Bcl-2. Photomicrographs of particular immunostaining of COX-2, p53, connexin-43, survivin, and Bcl-2 for fluorescence immunohistochemistry analyses shown by a greenish-yellow color and nuclear propidium iodide staining shown by a red color. Original magnification, 40×. Black arrows indicate immunopositive cells. Significant differences between Gp-I (control) and Gp-II (DSS) are shown by *** p < 0.001 and those between Gp-II and Gp-III are shown by the symbol # p < 0.05.

    Article Snippet: We employed the following primary antibodies: anti-rat p53 rabbit polyclonal antibody (Santa Cruz, dilution 1:100); anti-rat connexin-43 mouse monoclonal antibody (Santa Cruz, dilution 1:400); anti-rat COX-2 polyclonal antibody (Santa Cruz Biotechnology Inc., dilution 1:300); anti-rat survivin polyclonal antibody (Cell Signaling, dilution 1:300), and anti-rat Bcl-2 polyclonal antibody (Santa Cruz Biotechnology Inc., dilution 1:200).

    Techniques: Immunohistochemical staining, Staining, Immunostaining, Fluorescence, Immunohistochemistry, Control